Osteoporosis Reversal Program

Name: Osteoporosis Reversal Program
Brand: Your Holistic Doc
SKU: YHD-002-04
Price: 116.95 USD
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The difference between osteopenia and osteoporosis is that in osteopenia the bone loss is not as severe as in osteoporosis. That means someone with osteopenia is more likely to fracture a bone than someone with a normal bone density but is less likely to fracture a bone than someone with osteoporosis. Healthy Bone Turn-over Bone is a dynamic, living tissue which is continuously remodeled. As old bone is reabsorbed by osteoclasts, (cells that break bone down), the “old and stressed” bone is replaced with healthy new bone by osteoblasts (new bone cells). In a nutshell,...

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The difference between osteopenia and osteoporosis

is that in osteopenia the bone loss is not as severe as in osteoporosis. That means someone with osteopenia is more likely to fracture a bone than someone with a normal bone density but is less likely to fracture a bone than someone with osteoporosis.

Healthy Bone Turn-over

Bone is a dynamic, living tissue which is continuously remodeled. As old bone is reabsorbed by osteoclasts, (cells that break bone down), the “old and stressed” bone is replaced with healthy new bone by osteoblasts (new bone cells).

In a nutshell, a well balance bone support supplement, well balanced K2 and D3 5000 with more K2 and A is in my program, PLUS the addition of Strontium to address and reverse Osteoporosis.

The Connection between Osteoporosis and Atherosclerosis

Bridging the vessels and the bones

Have you ever wondered why so many aging people have calcified plaque in their arteries and yet walk around with brittle bones that are markedly depleted of calcium? Vitamin K2 is the link between these diseases.

•Since the 1970’s, we have been discovering many non-clotting Vitamin K2 dependent proteins found ubiquitously in the body, including the bones and the vessels.

Vitamin K2 deficiency is an epidemic in the country as is Vitamin D deficiency. Vitamin K2 availability decreases with aging.

Osteocalcin (OC or bone Gla protein, BGP) and matrix Gla protein (MGP) are 2 members of the growing family of vitamin K2-dependent (VKD) proteins found not to be involved in coagulation (blood clotting) and synthesized in the areas that they operate, such as the bones and the vessels.

Very dependent upon K2 to operate, Osteocalcin is primarily involved in bone metabolism. This protein binds calcium (once activated by Vitamin K2). Calcium from all over the body is then transported from the bloodstream into the bone matrix!

Matrix Gla protein (MGP) plays a crucial role in bone and vascular health, through the Inhibition of Vascular Calcification!

Vascular Health:

MGP is a potent inhibitor of vascular calcification, a process where calcium deposits build up in the arteries. It does this by binding to calcium ions and preventing their crystallization.

Bone Health:

MGP is also involved in bone health by regulating the activity of bone-forming cells (osteoblasts) and bone-resorbing cells (osteoclasts).

The production and activity of Osteocalcin and MGP are regulated by our K2 levels in the blood.

Low levels of K2 are associated with an increased risk of cardiovascular disease from vascular calcification and Osteoporosis.

In Vitamin K2 deficiency, not only is the important Vitamin K2-dependent protein, Osteocalcin underactive, BUT so is its partner, Matrix G1a protein (MGP). What this means is that in K2 deficiency, MGP, a very powerful inhibitor of arterial calcification is unable to function and because it facilitates the removal of calcium from vessels to be transported to where the calcium belongs, such as in our bones where the osteocalcin correctly deposits the calcium, our bone integrity is compromised in K2 deficiency and we develop calcified vessels, i.e. hardening of the arteries.

Menaquinones is the name given to the two forms of Vitamin K2, both MK-4 and MK-7 that are critical for bone strength and to inhibit calcified vessels. Before reviewing how to use K2, we need to learn why it is CRITICAL TO

Say “NO!” to Fosemax! Why? I will tell you why.

Turning to Healthy Bone Turn-over

Bone is a dynamic, living tissue that is continuously remodeled. As old bone is resorbed by osteoclasts, (cells that break bone down), the “old and stressed” bone is replaced with healthy new bone by osteoblasts (new bone cells).

DO WE WANT TO INHIBIT RESORPTION?!

Of course not! We don't want rubbery bones, we want strong bones!

Bisphosphonate drugs (such as Fosemax and Actonel) increase the Bone Mineral Density (BMD) of the bones at the expense of our bone quality. Such drugs reduce the activity of osteoclast through cell death. These drugs act as an osteoclast poison. This is how the drugs slow resorption of bone. As a result, the drugs maintain “older” bone with poorer architectural quality, that our osteoclasts WOULD HAVE removed to lay new bone cells down, making it impossible to continue to build and HAVE Strong bones. As old bone is NOT torn down, the bone is more brittle because new bone cell growth is placed on top of old bone, the QUALITY of the bone will naturally be diminished and weaker.

NOW let's turn to Strontium

Nature’s Bisphosphonate

“the way life should be” says your bones!

• The first vigorous studies were launched in Europe in the 1990’s to evaluate Strontium’s bone-building abilities. Large-scale, double-blind, randomized, placebo-controlled human studies were performed. All the studies showed statistically significant increased BMD and decreased fracture risk in both the spine as well as the hip with no reportable side effects.

How Strontium Works

Increases the replication of osteoblasts
DNA synthesis is increased in osteoblasts
Decrease the number of osteoclasts as well as their surface area covered
Does not kill osteosclasts, but slows their maturation

Result: Improved Quality and Quantity of Bone